MeSH: Dementia, Multi-Infarct - Finto

Haematologica, Volume 103, Issue 8 by Haematologica - issuu

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1 Despite its impact on the brain, there are currently no specific treatments for SVD, and therapeutic options for secondary prevention are particularly limited compared with those for other common causes of stroke. A multimodal MRI model explains a large proportion of the variation in executive function in cerebral small vessel disease. In particular, diffusion tensor imaging correlates best with executive function and is the most sensitive to change. This supports the use of MRI, in particular diffusion tenso … Small airway disease is defined as a pathologic condition in which the small conducting airways are affected either primarily or in addition to alveolar or interstitial lung changes. For the pathologist, small airway disease has the same meaning as bronchiolitis, a nonspecific term used to describe inflammation of the membranous and respiratory bronchioles. Small vessel disease is a disorder of cerebral microvessels that causes white matter hyperintensities and several other common abnormalities (eg, recent small subcortical infarcts and lacunes) seen on brain imaging. Despite being a common cause of stroke and vascular dementia, the underlying pathogenesis is poorly understood.

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This disease affects the cerebral vessels of small diameters. Cerebral small vessel disease (SVD) includes pathology of the brain's small arteries, arterioles, capillaries, venules, and veins.1 Key risk factors for SVD include age and cardiovascular risk factors,2,3 although vascular risk does not fully account for SVD, pointing to its multifactorial nature and, as of yet, unclear underlying mechanism. A multimodal MRI model explains a large proportion of the variation in executive function in cerebral small vessel disease. In particular, diffusion tensor imaging correlates best with executive function and is the most sensitive to change.

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Causes include atherosclerosis, inflammatory vasculitides, and noninflammatory vasculopathies such as postpartum angiopathy, cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL), Susac syndrome, and Sneddon syndrome.

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The latter correlates with inflammation and increased vascular permeabili Cerebral small vessel disease (SVD) is an umbrella term covering a variety of abnormalities related to small blood vessels in the brain. Because most brain tissue  Introduction. Vascular cognitive impairment is a common yet preventable cause for dementia. It needs high degree of suspicion and appropriate designing of  This damage may be caused by: high blood pressure high cholesterol obesity diabetes mellitus The treatment for small vessel disease involves medications to control the narrowing of your small blood vessels that could lead to a heart attack and to relieve pain.

Cerebral small vessel disease (SVD) includes pathology of the brain's small arteries, arterioles, capillaries, venules, and veins.1 Key risk factors for SVD include age and cardiovascular risk factors,2,3 although vascular risk does not fully account for SVD, pointing to its multifactorial nature and, as of yet, unclear underlying mechanism. Small vessel disease (SVD) of the brain accounts for ≈25% to 30% of strokes and is a leading cause of age-related and hypertension-related cognitive decline and disability. 1 Despite its impact on the brain, there are currently no specific treatments for SVD, and therapeutic options for secondary prevention are particularly limited compared with those for other common causes of stroke. A multimodal MRI model explains a large proportion of the variation in executive function in cerebral small vessel disease.
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Small vessel disease is a risk factor of ischemic or hemorrhagic stroke and a direct cause of lacunar and hemorrhagic infarcts. Moreover, it is associated of cognitive impairment, like psychomotor retardation, deficits of attention, planning, and set-shifting, and dysexecutive syndrome. This disease affects the cerebral vessels of small diameters. Cerebral small vessel disease (SVD) includes pathology of the brain's small arteries, arterioles, capillaries, venules, and veins.1 Key risk factors for SVD include age and cardiovascular risk factors,2,3 although vascular risk does not fully account for SVD, pointing to its multifactorial nature and, as of yet, unclear underlying mechanism. A multimodal MRI model explains a large proportion of the variation in executive function in cerebral small vessel disease.

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Changes to these vessels can damage white matter — the brain tissue that contains Se hela listan på gbs-cidp.org Multifocal motor neuropathy is a rare disorder characterized by slowly progressive muscle weakness, primarily of the arms and legs. The disorder is considered to be immune-mediated, which means there is inflammation resulting from abnormal functioning of the immune system and the presence of specific autoantibodies that target a specific protein in the body. Disease definition. A small vessel vasculitis characterized by neutrophilic inflammation predominantly limited to the superficial cutaneous postcapillary venules and without systemic vasculitis or glomerulonephritis. Typical presentation is of unifocal or multifocal palpable purpura on the lower extremities. In contrast, the dark spots contained both clotted and leaky blood vessels but no immune response. “We were completely surprised.

1 Despite its impact on the brain, there are currently no specific treatments for SVD, and therapeutic options for secondary prevention are particularly limited compared with those for other common causes of stroke. Small vessel disease is a risk factor of ischemic or hemorrhagic stroke and a direct cause of lacunar and hemorrhagic infarcts. Moreover, it is associated of cognitive impairment, like psychomotor retardation, deficits of attention, planning, and set-shifting, and dysexecutive syndrome. This disease affects the cerebral vessels of small diameters. Cerebral small vessel disease (SVD) includes pathology of the brain's small arteries, arterioles, capillaries, venules, and veins.1 Key risk factors for SVD include age and cardiovascular risk factors,2,3 although vascular risk does not fully account for SVD, pointing to its multifactorial nature and, as of yet, unclear underlying mechanism. A multimodal MRI model explains a large proportion of the variation in executive function in cerebral small vessel disease. In particular, diffusion tensor imaging correlates best with executive function and is the most sensitive to change.